Publications of Michael Naumann
All genres
Journal Article (29)
2009
Journal Article
8, 22 (2009)
Identification of novel Cyclooxygenase-2-dependent genes in Helicobacter pylori infection in vivo. Molecular Cancer 2005
Journal Article
386 (5), pp. 481 - 490 (2005)
The anti-inflammatory compound curcumin inhibits Neisseria gonorrhoeae-induced NF-kappa B signaling, release of proinflammatory cytokines/chemokines and attenuates adhesion in late infection. Biological Chemistry 2004
Journal Article
322 (3), pp. 860 - 866 (2004)
Analysis of the type IV secretion system-dependent cell motility of Helicobacter pylori-infected epithelial cells. Biochemical and Biophysical Research Communications
Journal Article
18 (1), pp. 218 - 220 (2004)
Helicobacter pylori stimulates host vascular endothelial growth factor-A (vegf-A) gene expression via MEK/ERK-dependent activation of Sp1 and Sp3. FASEB Journal 2003
Journal Article
5 (11), pp. 821 - 834 (2003)
Helicobacter pylori stimulates host cyclooxygenase-2 gene transcription: critical importance of MEK/ERK-dependent activation of USF1/-2 and CREB transcription factors. Cellular Microbiology
Journal Article
161 (2), pp. 249 - 255 (2003)
Helicobacter pylori CagA protein targets the c-Met receptor and enhances the motogenic response. Journal of Cell Biology 2002
Journal Article
277 (9), pp. 7438 - 7446 (2002)
Nuclear factor-kappa B directs carcinoembryonic antigen-related cellular adhesion molecule 1 receptor expression in Neisseria gonorrhoeae-infected epithelial cells. Journal of Biological Chemistry
Journal Article
16 (3), pp. 417 - 419 (2002)
B-Raf/Rap1 signaling, but not c-Raf-1/Ras, induces the histidine decarboxylase promoter in Helicobacter pylori infection. FASEB Journal 2001
Journal Article
61 (23), pp. 8416 - 8421 (2001)
The constitutive photomorphogenesis 9 signalosome directs vascular endothelial growth factor production in tumor cells. Cancer Research
Journal Article
291 (4), pp. 299 - 305 (2001)
Host cell signaling in Helicobacter pylori infection. International Journal of Medical Microbiology
Journal Article
276 (26), pp. 24331 - 24340 (2001)
Pathogenic Neisseria trigger expression of their carcinoembryonic antigen-related cellular adhesion molecule 1 (CEACAM1; previously CD66a) receptor on primary endothelial cells by activating the immediate early response transcription factor, nuclear factor-kappa B. Journal of Biological Chemistry
Journal Article
40 (4), pp. 815 - 823 (2001)
Pathogenicity island-dependent activation of Rho GTPases Rac1 and Cdc42 in Helicobacter pylori infection. Molecular Microbiology
Journal Article
276 (1), pp. 804 - 810 (2001)
Helicobacter pylori-induced prostaglandin E-2 synthesis involves activation of cytosolic phospholipase A(2) in epithelial cells. Journal of Biological Chemistry 2000
Journal Article
275 (50), pp. 39779 - 39785 (2000)
p21-activated kinase 1 activates the nuclear factor kappa B (NF-kappa B)-inducing kinase-I kappa B kinases NF-kappa B pathway and proinflammatory cytokines in Helicobacter pylori infection. Journal of Biological Chemistry
Journal Article
60 (8), pp. 1109 - 1114 (2000)
Nuclear factor-kappa B activation and innate immune response in microbial pathogen infection. Biochemical Pharmacology
Journal Article
2 (2), pp. 155 - 164 (2000)
Translocation of the Helicobacter pylori CagA protein in gastric epithelial cells by a type IV secretion apparatus. Cellular Microbiology
Journal Article
2 (2), pp. 155 - 164 (2000)
Translocation of the Helicobacter pylori CagA protein in gastric epithelial cells by a type IV secretion apparatus. Cellular Microbiology
Journal Article
275 (5), pp. 3629 - 3636 (2000)
Helicobacter pylori activates the histidine decarboxylase promoter through a mitogen-activated protein kinase pathway independent of pathogenicity island-encoded virulence factors. Journal of Biological Chemistry 1999
Journal Article
274 (50), pp. 35297 - 35300 (1999)
COP9 signalosome-directed c-Jun activation/stabilization is independent of JNK. Journal of Biological Chemistry
Journal Article
274 (44), pp. 31655 - 31662 (1999)
Activation of activator protein 1 and stress response kinases in epithelial cells colonized by Helicobacter pylori encoding the cag pathogenicity island. Journal of Biological Chemistry